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Morphologic alterations in the SV, a key structure for producing and maintaining the endocochlear potential (EP), decrease the EP and consequently, affect the cochlear amplification of acoustic signals leading to an increase in auditory thresholds, even in the absence of hair cell death (Gates and Mills, 2005

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The increases in TGF1 protein expression and SMAD3 phosphorylation induced by high glucose were significantly blocked in PSCs treated with GSH (Figs

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