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Hong J, Wang L, Zhang T, Zhang B, Chen L

Metabolic regulation-mediated reversion of the tumor immunosuppressive microenvironment for potentiating cooperative metabolic therapy and immunotherapy

Thus, CXCL1 and its receptors may be novel targets for therapeutic intervention to prolong PD therapy (Table 2

Targeting intrinsic dependency on ser/gly synthesis synergizes with RT, inhibiting cell proliferation and clonogenicity of NSCLC cell lines Since NSCLC cells seem to be dependent on ser/gly metabolism to recover and survive from RT induced cellular damage, we aimed to investigate whether these NSCLC cells are more sensitive to the combination of RT and ser/gly metabolism-targeting therapy