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Description

who attributed ACs effect against OSCC to methyltransferase-like 3 (METTL3) downregulation, which in turn disrupts N6-methyladenosine (m6A)-dependent processing of miR-31-5p, leading to enhanced homeodomain-interacting protein kinase 2 (HIPK2) activity and reinforcing AC's inhibitory effects on OSCC cell behavior (Huang et al

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Cell Physiol Biochem 37(4):12401256

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Res Synth Methods 3

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"Non-enzymatic lipid oxidation products in biological systems: assessment of the metabolites from polyunsaturated fatty acids" (PDF)

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