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For instance, we have identified a confirmed target pathway for anti-fungal drugs with glutathione metabolism (GSH), attributed to fungal mitochondrial maintenance, preservation of membrane integrity, regulation of transcription factors in stress response and protection against reactive oxygen species

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Current research suggests anodal tDCS induces a subthreshold, polarity-dependent membrane polarization that induces neural plasticity via N-methyl-D-aspartate, gamma-aminobutyric acid, brain-derived neurotrophic factor, and calcium-dependent mechanisms [76, 274,275,276]