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These approaches are subject to many of the same limitations as nutritional/microbiota therapies for TBI, including the lack of a suitable animal model of the human microbiome, heterogeneity in the methods/quality of microbiome data collection, the protracted timeline and inevitable presence of confounding variables in human diet trials [130]

In situations of stress, illness, or injury, the demand for glutamine by gut cells increases significantly to support repair mechanisms and prevent atrophy of the intestinal lining

Inflammation as a neurobiological mechanism of cognitive impairment in psychological stress

Long-term safety, efficacy, and patient-specific responses should be key focuses of future investigations