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The formation of metal-flavonoid complexes significantly enhances their bioavailability, positioning these compounds as promising candidates for next-generation therapeutics ( Ehretia tinifolia extract (ETME) counteracts this response by activating the protective Nrf2/HO-1 pathway while suppressing NF-B/MAPK-mediated inflammation

Based on our current understanding and findings, we summarized how fructose protects against acetaminophen-induced liver damage: (1) by increasing the expression of antioxidant genes through the ChREBP-FGF21 axis and (2) by regulating factors that are involved in the metabolism of APAP, such as CYP2E1 and glutathione [Figure 2]

Oxidative Stress: A Convergent Amplifier of Biomarker Dysregulation Oxidative stress in CIRS is not merely a downstream effect of inflammation but functions as a cross-cutting disruptor of cellular, mitochondrial, and genomic stability

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