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10.1096/fj.201600435RR 57

Semax (BDNF/TrkB pathway, neuroplasticity) and the Selank (GABAergic pathway, anxiolysis) act through non-competitive and complementary mechanisms

This activity may be driven by increased levels of brain-derived neurotrophic factor (BDNF) or through a more direct modulation of specific receptors that govern cognitive processes