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Redox dysregulation is believed to play a crucial role in the pathophysiology of schizophrenia, with reduced levels of the antioxidant GSH in the brain linked to oxidative stress and schizophrenia (20)

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Genetic polymorphism in the genes ABCB1 , ABCC1 , ACTG1 , CAPG , MAP4 , SLC5A7 , TTPA and TUBB1 could modulate the risk of developing neuropathy in children following vincristine treatment (Wright et al., 2019)