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OMIM 266130) is a rare autosomal-recessive metabolic disorder characterized by decreased glutathione (GSH) levels owing to defects in glutathione synthetase (GSS)

NFAT5 repressed NIK/NF-B2 pathway via ubiquitination and degradation of NIK by Akt1/cIAP1/2 We used the STRING database to identify the mediator molecule between NFAT5 and NIK/NF-B2

Lets dive into why this powerhouse nutrient is creating waves in wellness circles and how it can help you reclaim your vitality

Its pathological features include high inflammatory and immune response, oxidative stress, defective antioxidant defense system, altered protein homeostasis, cellular senescence, and apoptosis